As if these tentacled wonders weren’t already alien enough, the octopus' three hearts and blue copper-rich blood seals the deal.Octopuses have three hearts: one pumps blood around the body; the other two pump blood to the gills. ","Diagnostic criteria for takotsubo syndrome: A call for consensus","Transient midventricular ballooning syndrome: a new variant","Takotsubo cardiomyopathy or broken heart syndrome: A review article","The clinical features of takotsubo cardiomyopathy","BBC NEWS | Health | Medics 'can mend a broken heart,"Cardiomyopathy, Takotsubo Syndrome (Transient Apical Ballooning, Stress-Induced Cardiomyopathy, Gebrochenes-Herz Syndrome)","Broken Heart Syndrome – National Heart, Lung, and Blood Institute","Clinical management of takotsubo cardiomyopathy","Management and prognosis of stress (takotsubo) cardiomyopathy","Takotsubo Cardiomyopathy: A New Form of Acute, Reversible Heart Failure","Understanding Heartbreak: From Takotsubo to Wuthering Heights",Arrhythmogenic right ventricular dysplasia,https://en.wikipedia.org/w/index.php?title=Takotsubo_cardiomyopathy&oldid=975468839,Creative Commons Attribution-ShareAlike License,Schematic representation of takotsubo cardiomyopathy (A) compared to a normal heart (B),This page was last edited on 28 August 2020, at 18:02. One of the first sets of guidelines was initially published in 2004 and again in 2008 by the,Transient apical ballooning syndrome or takotsubo cardiomyopathy is found in 1.7–2.2% of patients presenting with,The diagnosis of takotsubo cardiomyopathy may be difficult upon presentation. ).Haemocyanin is less efficient than haemoglobin as an oxygen transporter. as.In short, the main criteria for the diagnosis of takotsubo cardiomyopathy are: the patient must have experienced a stressor before the symptoms began to arise; the patient's ECG reading must show abnormalities from a normal heart; the patient must not show signs of coronary blockage or other common causes of heart troubles; the levels of cardiac enzymes in the heart must be elevated or irregular; and the patient must recover complete contraction and be functioning normally in a short amount of time.Left ventriculography during systole showing apical ballooning akinesis with basal hyperkinesis in a characteristic takotsubo ventricle,Echocardiogram showing the effects of the disease.While medical treatments are important to address the acute symptoms of takotsubo cardiomyopathy, further treatment includes lifestyle changes.Although the symptoms of takotsubo cardiomyopathy usually go away on their own and the condition completely resolves itself within a few weeks, some serious short and long-term complications can happen that must be treated.For people with cardiogenic shock, medical treatment is based on whether a.Despite the grave initial presentation in some of the patients, most of the patients survive the initial acute event, with a very low rate of in-hospital mortality or complications. The pathophysiology is not well understood, but a sudden massive surge of.Takotsubo cardiomyopathy occurs worldwide.It occurs more commonly in postmenopausal women.Although there have been documented cases of TTS without a triggering stressor, it is widely recognized that TTS is preceded by a stressful event.Women, specifically postmenopausal women, are at greatest risk of developing TTS.It is currently being investigated if certain genetic traits associated with catecholamine receptors found on cardiac muscle cells play a role in the development of TTS.The typical presentation of takotsubo cardiomyopathy is chest pain with or without shortness of breath and associated.Stress is the main factor in takotsubo cardiomyopathy, with more than 85% of cases set in motion by either a physically or emotionally stressful event that prefaces the start of symptoms.The cause of takotsubo cardiomyopathy is not fully understood, but several mechanisms have been proposed.It is likely that there are multiple factors at play that could include some amount of vasospasm and failure of the microvasculature. The syndrome reached international audiences through the media in 2005 when the,Sudden temporary weakening of the heart muscle,CS1 maint: multiple names: authors list (,Sharkey, S., Lesser, J., & Maron, B. American Heart Association. (2011). It also affects far more women than men with 90% of cases being women, most postmenopausal. The mantle is a highly muscled structure that houses all of the animal's organs. More cases of the syndrome appeared in Japan within the next decade, although western medicine had still not acknowledged it. ","Apical ballooning of the left ventricle: first series in white patients","Tako-Tsubo cardiomyopathy: new insights into the possible underlying pathophysiology","Takotsubo Cardiomyopathy Triggered by Wasabi Consumption: Can Sushi Break Your Heart? The largest specimen of this species to be scientifically documented was an animal with a live mass of 71 kg (156.5 lb). The giant Pacific octopus has three hearts, nine brains and blue blood, making reality stranger than fiction. The Japanese at last reported about the syndrome to the west in 2001 under the name "transient LV apical ballooning" though at this point the west had already heard of numerous cases.

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